N	Concordance
1	lymphocytes responding to soluble HIV-1 antigen are not deleted in HIV-1-infecte
2	 Compared with the placebo group, PAI-1 antigen levels were 44% lower (P=.004) a
3	iated primarily by very late activation antigen-3, which leads to a subsequent i
4	ne can be preduced by overexpressing an antigen in cultured cells prior to purif
5	gnificance of elevated levels of cancer antigen 125 (CA125), placental alkaline 
6	, and for vimentin and carcinoembryonic antigen in some cases. No immunoreactivi
7	es displayed a lower expression of CD26 antigen. Comparing cytokine production a
8	ting signal transduction via the B cell antigen receptor (BCR). These PTPs diffe
9	g sepsis. The altered expression of Fas antigen and FasL may play a pivotal role
10	way may lead to the rare IP blood-group antigen and to globoside-like molecules 
11	stocompatibility complex (MHC) class II antigen expression. We interpret the pre
12	sly expressed beta 1 integrin very late antigen 5 is readily activated by PMA, w
13	 presence of proliferating cell nuclear antigen, a marker for mitogenic activity
14	ody-specific immobilization of platelet antigen assay. HLA antibodies were detec
15	cell lysates containing the recombinant antigen usually by affinity chromatograp
16	lebrand factor, and factor VIII-related antigen were monitored perioperatively. 
17	 partial inactivation to organ-specific antigen allows nondestructive thyroiditi
18	bination of specific anti-viral surface antigen effect and a nonspecific graft v
19	liferation. To accomplish this, large T antigen has to control the expression of
20	of cytokines that upregulate the target antigen or expand the effector populatio
21	wo epitopes derived from the same viral antigen and presented through the same M