N	Concordance
1	 both CD4+ and CD8+ cells with a type 2 cytokine phenotype (i.e. Th2 and Tc2 typ
2	e-chemotactic and neutrophil-activating cytokine, in the human preovulatory foll
3	ere analyzed for their ability to alter cytokine induction by target cell intera
4	recently emerged as an antiinflammatory cytokine that inhibits the secretion of 
5	s oncoproteins, inflammation-associated cytokine IL-6 and the viral X transactiv
6	lular vaccine: comparison of the T cell cytokine profiles in infants and mice.  
7	erleukin-6 (IL-6), the main circulating cytokine, is putatively a major mediator
8	ronment and abnormalities in endogenous cytokine production by neoplastic cells.
9	 M (OSM), a member of the hematopoietic cytokine family, has been implicated in 
10	terleukin 12 (IL-12) is a heterodimeric cytokine which promotes the development 
11	 presentation. A major immunomodulatory cytokine, gamma-interferon (gamma-IFN), 
12	 the production of an immunosuppressive cytokine, IL-10, might contribute to the
13	 different patterns of allergen-induced cytokine production. In vitro allergen-i
14	 are also observed at the intracellular cytokine level. Additional studies revea
15	lar IL-1 beta staining was due to local cytokine synthesis rather than protein a
16	of decreased in vitro murine macrophage cytokine release after exposure to carbo
17	ctor-alpha (TNF-alpha) is a pleiotropic cytokine which stimulates the synthesis 
18	s factor (TNF)-alpha, a proinflammatory cytokine with negative inotropic effects
19	t study examined whether maternal serum cytokine levels are useful for the diagn
20	 in vivo T cell activation and systemic cytokine release, and a human anti-mouse
21	ther, our results indicate that the Th2 cytokine IL-9 can be expressed by both I
22	rtant immunoregulatory component of the cytokine network in rheumatoid arthritis