N	Concordance
1	ich are then activated. These activated leukocytes release free radicals and pro
2	chronic renal failure in whom antihuman leukocyte antigen antibodies developed a
3	lls (granulocytes) and peripheral blood leukocytes (PBLs). Six of the 55 serum s
4	he readiness to help adhere circulating leukocytes exposed to proinflammatory st
5	ulation. These results suggest that CSF leukocytes are not primarily responsible
6	 induced lethal GVHD, but delayed donor leukocyte infusion (DLI) 5 days after TB
7	, capillary blood flow, and endothelial leukocyte adhesion were measured in endo
8	eloped a new method, called fluorescein leukocyte angiography (FLA), to display 
9	ere were even less patients with a high leukocyte count (15 vs 38%, P=0.003), a 
10	ecificities to human histocompatibility leukocyte antigen (HLA)-B35. Among the e
11	NS1 or NS2a proteins. Analysis of human leukocyte antigen (HLA) restriction reve
12	dogenous inhibitor of TNF-alpha-induced leukocyte chemotaxis via inhibition of I
13	ion of the amount of other inflammatory leukocytes in the CNS of these treated a
14	est (LST). Peripheral blood mononuclear leukocytes (PBM L) cultures were induced
15	Recent evidence suggests that passenger leukocytes migrate after organ transplan
16	ophage activation and polymorphonuclear leukocyte recruitment patterns and to co
17	shing liver graft tolerance, DA splenic leukocytes were further purified into th
18	cutoffs of 5%, 9%, and 10% of the total leukocyte count as an indicator of the d