N	Concordance
1	 mononuclear cells (PBMC). In addition, methylprednisolone (MP) was shown to be 
2	sporine (group II), or cyclosporine and methylprednisolone for evidence of acute
3	rculating lymphocyte subsets induced by methylprednisolone pulse therapy (MPT) a
4	iation following pulse cyclophosphamide/methylprednisolone treatment of multiple
5	carmustine, procarbazine, and high-dose methylprednisolone as additions to surge
6	s (group 1) were randomised to be given methylprednisolone (30 mg/kg) and 15 pat
7	ts receiving either monthly intravenous methylprednisolone (MP), intravenous cyc
8	tis) underwent hip injection with 80 mg methylprednisolone and lignocaine under 
9	 consistent with TON. A third course of methylprednisolone again led to improved
10	ATP supply. By comparing the effects of methylprednisolone with those of myxothi
11	(MP), intravenous cyclophosphamide plus methylprednisolone (CY/MP), methotrexate
12	ed significantly (P < 0.03) after pulse methylprednisolone treatment, in paralle
13	, 13 could not be biopsied, 16 received methylprednisolone, and 11 received anti
14	r intraarticular injection of synthetic methylprednisolone acetate. Immediately 
15	 tobramycin sulfate in combination with methylprednisolone acetate or dexamethas