N	Concordance
1	opic dermatitis may reflect an abnormal monocyte maturation/differentiation stat
2	(DC) as in lipopolysaccharide-activated monocytes, but intensively in CD40-activ
3	fluid, spleen, and lung. Newly adherent monocytes expressed CCR5 in all seven pa
4	ccompanied by increased granulocyte and monocyte counts, increased rectal temper
5	3 complex in the presence of autologous monocytes induces significantly greater 
6	od), the authors found that human blood monocytes actively bind labeled tubercul
7	noid biosynthesis in HPS patients CD14+ monocytes were isolated from plasma samp
8	ious cytokines, including the chemokine monocyte chemoattractant protein-1 (MCP-
9	ressed the question whether circulating monocytes show altered chemotaxis to MCP
10	oduction was investigated with cultured monocytes isolated from human peripheral
11	e role of cytokines such as granulocyte-monocyte colony stimulating factor (GM-C
12	ring these preparations with homologous monocytes, purified from PBMC preparatio
13	feration and lipopolysaccharide-induced monocyte production of proinflammatory c
14	posed mononuclear cells or HIV-infected monocyte/macrophages (M/Ms) is the princ
15	ibitor, did not affect the LPL-mediated monocyte binding. To assess whether spec
16	previously the glomerular expression of monocyte chemoattractant protein-1 (MCP-
17	t-induced lipidosis in human peripheral monocytes in vitro, as well as in a mono
18	In summary, the depletion of phagocytic monocytes from PSC products restores the
19	 that HIV-1-infected, activated primary monocytes can migrate between epithelial