N	Concordance
1	y suppressing inflammatory cytokine and monokine production in the vaginal mucos
2	s isolated from human peripheral blood. Monokine concentrations were determined 
3	N-gamma) was also induced indirectly by monokines. Collectively, our results ind
4	r regulation of A beta PP metabolism by monokines may facilitate vascular amyloi
5	ed expression of mRNA specific for each monokine on exposure of monocytes to FN.
6	emopoietic suppressive effects of human monokine induced by IFN-gamma (HuMig), a
7	lls of the predominant pro-inflammatory monokine TNF-alpha and changes were also
8	factor alpha (TNF alpha), a pleiotropic monokine, is of special interest because
9	lls. These results suggest that several monokines are active in regulating the t
10	tion pathways involved in FN-stimulated monokine secretion. FN-stimulated TNF-al
11	sted because they stimulated a stronger monokine release. T cell proliferation a